Results of national registration of familial adenomatous polyposis
- Diminutive adenomas were markers of more neoplasia. Roberts, John; Greenberger, Norton J. // Annals of Internal Medicine;08/15/98, Vol. 129 Issue 4, p314
Presents a summary of the article `Importance of Adenomas 5 mm or Less in Diameter That are Detected by Sigmoidoscopy,' by T.E. Read, J.D. Read and L.F. Butterfly, which was published in the 1997 edition of the periodical `New England Journal of Medicine.'
- Challenge in the differentiation between attenuated familial adenomatous polyposis and hereditary nonpolyposis colorectal cancer: case report with review of the literature. Cao, Yangming; Pieretti, Maura; Marshall, Jay; Khattar, Nada H.; Chen, Bifen; Kam-Morgan, Lauren; Lynch, Henry // American Journal of Gastroenterology;Jul2002, Vol. 97 Issue 7, p1822
The clinical differentiation between hereditary nonpolyposis colorectal cancer (HNPCC) and attenuated familial adenomatous polyposis (AFAP) is very difficult. The 62-yr-old proband presented with duodenal adenocarcinoma. His history of subtotal colectomy for colon cancer, the rarity of duodenal...
- Familial adenomatous polyposis. Church, James // American Journal of Gastroenterology;Feb2007, Vol. 102 Issue 2, p462
A letter to the editor is presented in response to article about familial adenomatous polyposis.
- COX 2 inhibitors might be useful in cancer prevention. Gottlieb, Scott // BMJ: British Medical Journal (International Edition);10/30/99, Vol. 319 Issue 7218, p1155
Reports on the treatment of familial adenomatous polyposis. Discussion on familial adenomatous polyposis; Overview of a study on patients with familial adenomatous polyposis; Details on the drug Celecoxib.
- Desmoids in familial adenomatous polyposis are monoclonal proliferations. Middleton, S B; Frayling, I M; Phillips, R K S // British Journal of Cancer;2/15/2000, Vol. 82 Issue 4, p827
Desmoids are poorly-understood, locally aggressive, non-metastasizing fibromatoses that occur with disproportionate frequency in patients with familial adenomatous polyposis (FAP). Their nature is controversial with arguments for and against a neoplastic origin. Neoplastic proliferations are by...
- Hyperparathyroidism and the general paediatrician. Cheetham, Tim; Bliss, Richard // Archives of Disease in Childhood;Oct2015, Vol. 100 Issue 10, p904
The authors discuss the significance of treating children with hyperparathyroidism for paediatricians. They state that diagnosis of the disease has become easier due to availability of biochemical tests, and mentions questions arising among paediatricians regarding the cause of the disease in...
- Multiple endocrine neoplasia in an orthodontic patient. Collins, Joanne M.; Chaudhry, Shahid I.; Gill, Daljit S.; Porter, Stephen R. // Journal of the American Dental Association (JADA);Oct2012, Vol. 143 Issue 10, p1093
The article presents a case of Multiple endocrine neoplasia, type 2B (MEN 2B), which was treated successfully for metastatic medullary thyroid cancer. It states that MEN 2B might culminate into conditions such as pheochromocytoma. Dental professionals could recognize the disorder, because MEN 2B...
- Identification and proposal of a potentially new clonal complex that is a common cause of MenB disease in central and eastern Canada. // Canadian Journal of Microbiology;Oct2012, Vol. 58 Issue 10, p1236
This study examined serogroup B meningococci (MenB) from invasive meningococcal disease (IMD) cases in the provinces of QuÃ©bec and Ontario in the last decade by multilocus sequence typing (MLST) to determine their sequence types (STs) and clonal complexes (CCs). Forty isolates from individual...
- Secretin-Stimulated Serum Gastrin Levels in Hyperparathyroid Patients from Families with Multiple Endocrine Adenomatosis Type I. Lamers, Cornelis B.; Buis, Jan T.; van Tongern, Jan // Annals of Internal Medicine;Jun77, Vol. 86 Issue 6, p719
Presents a study that determined the secretin-stimulated serum gastrin levels in hyperparathyroid patients from families with multiple endocrine adenomatosis. Secretin provocation testing; Methods used in the study; Findings.
- Five Families with Multiple Endocrine Adenomatosis. Snyder III, Ned; Scurry, Murphy T.; Deiss Jr., William P. // Annals of Internal Medicine;Jan72, Vol. 76 Issue 1, p53
Presents a study that screened families for involvement with multiple endocrine adenomatosis. Number of families with hypercalcemia; Detection of pancreatic tumors as the second commonest abnormality in the families; Evaluation of serum calcium of the families.