TITLE

A normal-calcium, low-protein, low-salt diet reduced recurrence of renal stones over 5 years

AUTHOR(S)
Curhan, Gary
PUB. DATE
September 2002
SOURCE
ACP Journal Club;Sep/Oct2002, Vol. 137 Issue 2, p62
SOURCE TYPE
Academic Journal
DOC. TYPE
Article
ABSTRACT
Presents a study that investigated the efficacy of a diet containing a normal amount of calcium but reduced amount of animal protein and salt versus a traditional low-calcium diet in men with recurrent calcium oxalate stones and hypercalciuria. Study design; Description of patients; Analysis of recurrence of symptomatic renal stone.
ACCESSION #
7519819

 

Related Articles

  • Are Phosphatonins Relevant in the Pathogenesis of Nephrolithiasis? Shaikh, Aisha; Kumar, Rajiv // AIP Conference Proceedings;2007, Vol. 900 Issue 1, p170 

    Hypercalciuria is the single most important risk factor for stone disease and renal phosphate excretion is a recognized cause of absorptive hypercalciuria. The recent identification of “phosphatonins” that increase renal phosphate excretion has raised the possibility that these...

  • Idiopathic Calcium Nephrolithiasis And Hypercalciuria: The Role Of Genes. Gambaro, Giovanni; Abaterusso, Cataldo // AIP Conference Proceedings;2007, Vol. 900 Issue 1, p110 

    Idiopathic calcium nephrolithiasis and hypercalciuria are multifactorial disease conditions, the pathogenesis of which involves the interaction of environmental and individual factors. Data support a strong role of genes in the pathogenesis of these two conditions. Findings obtained in monogenic...

  • Calciuric response to an acute acid load in healthy subjects and hypercalciuric calcium stone formers. Houillier, Pascal; Normand, Michel; Froissart, Marc; Blanchard, Anne; Jungers, Paul; Paillard, Michel // Kidney International;Sep1996, Vol. 50 Issue 3, p987 

    Excessive animal protein consumption is associated with a greater risk of occurrence of renal calcium stone, presumably because of the attendant endogenous acid production. Indeed, chronic acid load enhances urinary calcium excretion possibly through an increased bone calcium release. Because...

  • 189: VDR Apa-1 gene polymorphism in nephrolithiasis. Panigrahy, Benu; Singh, Shrawan K.; Khullar, Madhu; Kumar, Santosh; Acharya, Naveen; Agarwal, Mayank; Mandal, Arup K. // Indian Journal of Urology;Supplement2, Vol. 24, pS116 

    Introduction: To find out the association of VDR gene polymorphism with respect to Apa 1, in nephrolithiasis and the familial pattern of inheritance of nephrolithiasis. Methods: 130 patients with nephrolithiasis and 100 controls were studied. They were evaluated clinically and biochemically. The...

  • Biochemical characterization of primary hyperparathyroidism with and without kidney stones. Odvina, Clarita V.; Sakhaee, Khashayar; Heller, Howard J.; Peterson, Roy D.; Poindexter, John R.; Padalino, Paulette K.; Pak, Charles Y. C. // Urological Research;Jun2007, Vol. 35 Issue 3, p123 

    The exact metabolic-physiological background for kidney stone formation in primary hyperparathyroidism (PHPT) is unclear. To obtain clarification, this retrospective data analysis was conducted in 131 patients with PHPT who had undergone a detailed ambulatory evaluation on a random diet since...

  • Defective renal calcium reabsorption in genetic hypercalciuric rats. Tsuruoka, Shuichi; Bushinsky, David A.; Schwartz, George J. // Kidney International;May1997, Vol. 51 Issue 5, p1540 

    Idiopathic hypercalciuria is a frequent cause of calcium (Ca) containing kidney stones. We have previously shown that there is increased intestinal Ca absorption in selectively inbred genetic hypercalciuric stone forming (GHS) rats; however, excess Ca excretion persists when the rats are fed a...

  • R990G polymorphism of calcium-sensing receptor does produce a gain-of-function and predispose to primary hypercalciuria. Vezzoli, G.; Terranegra, A.; Arcidiacono, T.; Biasion, R.; Coviello, D.; Syren, M. L.; Paloschi, V.; Giannini, S.; Mignogna, G.; Rubinacci, A.; Ferraretto, A.; Cusi, D.; Bianchi, G.; Soldati, L. // Kidney International;Jun2007, Vol. 71 Issue 11, p1155 

    An association between the R990G polymorphism of the CaSR gene, coding for calcium-sensing receptor, and primary hypercalciuria was found in kidney stone formers. To confirm this relationship, we investigated hypercalciuric women without stones and studied the effect of CaSR gene in human...

  • Pathophysiology of the Hypercalciuria in the Genetic Hypercalciuric Stone-Forming Rats. Bushinsky, David A. // AIP Conference Proceedings;2007, Vol. 900 Issue 1, p118 

    Given evidence for a genetic cause of hypercalciuria, we screened adult male and female Sprague-Dawley (SD) rats for hypercalciuria and used those with the highest urinary calcium excretion to breed the next generation, followed by subsequent selection and inbreeding of their most hypercalciuric...

  • Male hypercalciuric stone formers with low renal calcium reabsorption. Walker, Valerie; Cook, Paul; Griffin, Damian G. // Journal of Clinical Pathology;Apr2014, Vol. 67 Issue 4, p355 

    Aims Hypercalciuria is a common poorly understood abnormality among stone formers. We aimed to identify hypercalciuric male stone formers with a primary defect in renal calcium reabsorption and to look for associated risk factors. Methods A retrospective cross-sectional database study of 623...

Share

Read the Article

Courtesy of VIRGINIA BEACH PUBLIC LIBRARY AND SYSTEM

Sorry, but this item is not currently available from your library.

Try another library?
Sign out of this library

Other Topics