TITLE

Attenuation of ischaemia reperfusion injury by human thioredoxin

AUTHOR(S)
Fukuse, T; Hirata, T; Yokomise, H; Hasegawa, S; Inui, K; Mitsui, A; Hirakawa, T; Hitomi, S; Yodoi, J; Wada, H
PUB. DATE
April 1995
SOURCE
Thorax;Apr1995, Vol. 50 Issue 4, p387
SOURCE TYPE
Academic Journal
DOC. TYPE
Article
ABSTRACT
No abstract available.
ACCESSION #
66066480

 

Related Articles

  • The Therapeutic Role of Taurine in Ischaemia-Reperfusion Injury. Kingston, R.; Kelly, C. J.; Murray, P. // Current Pharmaceutical Design;Jul2004, Vol. 10 Issue 19, p2401 

    As a non-toxic endogenous antioxidant, the semi-essential amino acid taurine is a potential attenuator of oxidative damage such as that produced by ischaemia-reperfusion injury. Ischaemia-reperfusion injury is a well established if paradoxical phenomenon whereby ischaemic tissue, doomed to...

  • High glucose sensitizes adult cardiomyocytes to ischaemia/reperfusion injury through nitrative thioredoxin inactivation. Ronghua Luan; Shaowei Liu; Tao Yin; Wayne Bond Lau; Qiong Wang; Wenyi Guo; Haichang Wang; Ling Tao // Cardiovascular Research;Jul2009, Vol. 83 Issue 2, p294 

    : Aims Ischaemic cardiac injury is significantly increased in diabetic patients, but its underlying mechanisms remain incompletely understood. The current study attempted to identify new molecular mechanisms potentially contributive to hyperglycaemic-exaggeration of myocardial ischaemic injury. ...

  • Protective effects of sphingosine-1-phosphate receptor agonist treatment after myocardial ischaemia–reperfusion. Ulrich Hofmann; Natalie Burkard; Carolin Vogt; Annemarie Thoma; Stefan Frantz; Georg Ertl; Oliver Ritter; Andreas Bonz // Cardiovascular Research;Jul2009, Vol. 83 Issue 2, p285 

    : Aims Several experimental studies have demonstrated protection against cardiac ischaemia–reperfusion injury achieved by pre-treatment with exogenous sphingosine-1-phosphate (S1P). We tested the hypothesis that pharmacological S1P receptor agonists improve recovery of function when...

  • INTERMEDIN VIA AM1-RECEPTOR SIGNALLING PROTECTS HUMAN VASCULAR AND CARDIAC NON-VASCULAR CELLS FROM ISCHAEMIA-REPERFUSION INJURY. Ferguson, M.; Campbell, M.; Donaghy, L.; McAleer, S.; ÒRegan, A.; Harbinson, M.; Bell, D. // Heart;Nov 2012 Supplement, Vol. 98, pA6 

    An abstract of the article "Intermedin Via Ami-Receptor Signalling Protects Human Vascular and Cardiac Non-Vascular Cells from Ischaemia-Reperfusion Injury," by M. Ferguson and colleagues is presented.

  • REMOTE ISCHAEMIC PRECONDITIONING (rIPC) REDUCES CARDIAC AND PULMONARY DYSFUNCTION FOLLOWING EXPERIMENTAL CARDIOPULMONARY BYPASS. Kharbanda, R.; Li, J.; Konstantinov, I.; M. Cheung; White, P.; Frndova, H.; Stokoe, J.; Cox, P.; Vogel, M.; van Arsdell, G.; MacAllister, R.; Redington, A. // Heart;May2004 Supplement 2, Vol. 90, pA2 

    This article focuses on a study according to which remote ischaemic preconditioning (RIPC) reduces cardiac and pulmonary dysfunction following experimental cardiopulmonary bypass. RIPC reduces injury caused by ischaemia reperfusion in distant organs. Cardiopulmonary bypass (CPB) is associated...

  • Neuroprotective effects of tramadol on cerebral injuries caused by hind limb ischaemia/reperfusion in rats. Takhtfooladi, Mohammad; Jahanshahi, Amirali; Sotoudeh, Amir; Daneshi, Mohammad; Aslani, Kimia; Takhtfooladi, Hamed // Comparative Clinical Pathology;Sep2014, Vol. 23 Issue 5, p1141 

    Skeletal muscle ischaemia-reperfusion-induced acute remote injury is mediated by activated neutrophils and formation of free radicals. Several investigators have demonstrated that the opioid pathway is involved in tissue preservation during hypoxia or ischaemia. Tramadol hydrochloride is an...

  • Poster presentation 2002. Sutton, Steven W.; Patel, Amit N.; Schmidt, Lori A.; Hunley, Eric K.; Yancey, Lloyd W.; Hebeler, Robert F.; Cheung, Edson H.; Henry III, A. Carl; Hamman, Baron L.; Wood, Richard E.; Samankatiwat, Piya; Chen, Q.; Shepard, S.; Clinton, V. // Perfusion;Jan2003 Supplement, Vol. 18, p89 

    Presents information on cardioplegic leukocyte depletion during cardiopulmonary bypass. Benefits of leukocyte filters for improved oxygenation and decreased length of hospital stay; Method for attenuating myocardial ischaemia reperfusion injury; Reduction in activated neutrophils by...

  • Activation of hypoxia-inducible factors ameliorates hypoxic distal tubular injury in the isolated perfused rat kidney. Rosenberger, Christian; Rosen, Seymour; Shina, Ahuva; Frei, Ulrich; Eckardt, Kai-Uwe; Flippin, Lee A.; Arend, Michael; Klaus, Stephen J.; Heyman, Samuel N. // Nephrology Dialysis Transplantation;Nov2008, Vol. 23 Issue 11, p3472 

    Background. Preconditional activation of HIF with specific prolyl-hydroxylase inhibitors (PHD-I) attenuates proximal tubular injury, induced by warm ischaemia/ reperfusion (Bernhardt, JASN, 2006). Distal tubular damage occurs in humans with acute kidney injury (AKI), in experimental contrast...

  • The antioxidant EPC-K1 ameliorates brain injury by inhibiting lipid peroxidation in a rat model of transient focal cerebral ischaemia. Kato, N.; Yanaka, K.; Nagase, S.; Hirayama, A.; Nose, T. // Acta Neurochirurgica;Jun2003, Vol. 145 Issue 6, p489 

    Summary¶Background. Cerebral ischaemia-reperfusion injury is associated with the generation of reactive oxygen species during the early phases of reoxygenation. EPC-K1, a phosphate diester of vitamins C and E, has been reported to possess potent hydroxyl radical scavenging activity. This...

  • Ischaemia Reperfusion Injury (Book Review). Bell, PRF // Journal of the Royal College of Surgeons of Edinburgh;Jun99, Vol. 44 Issue 3, p209 

    Reviews the book 'Ischaemia Reperfusion Injury,' edited by Pierce A. Gace and Robert T. Mathie.

Share

Read the Article

Courtesy of VIRGINIA BEACH PUBLIC LIBRARY AND SYSTEM

Sign out of this library

Other Topics