Coral Disease in a Warmer World
Tags: OCEAN temperature; DISEASE susceptibility; CORALS -- Diseases; NATURAL immunity; CORAL declines
Related Articles
- Natural Disease Resistance in Threatened Staghorn Corals. Vollmer, Steven V.; Kline, David I. // PLoS ONE;2008, Vol. 3 Issue 11, p1
Disease epidemics have caused extensive damage to tropical coral reefs and to the reef-building corals themselves, yet nothing is known about the abilities of the coral host to resist disease infection. Understanding the potential for natural disease resistance in corals is critically important,...
- Coral reef countdown. // New Scientist;4/9/2011, Vol. 210 Issue 2807, p6
The article reports that if the rate of carbon dioxide (CO2) released into the atmosphere continues unabated, ocean warming will occur despite human intervention to stop it and threaten the survival of coral reefs.
- Temperature regulation of virulence factors in the pathogen Vibrio coralliilyticus. Kimes, Nikole E; Grim, Christopher J; Johnson, Wesley R; Hasan, Nur A; Tall, Ben D; Kothary, Mahendra H; Kiss, Hajnalka; Munk, A Christine; Tapia, Roxanne; Green, Lance; Detter, Chris; Bruce, David C; Brettin, Thomas S; Colwell, Rita R; Morris, Pamela J // ISME Journal: Multidisciplinary Journal of Microbial Ecology;Apr2012, Vol. 6 Issue 4, p835
Sea surface temperatures (SST) are rising because of global climate change. As a result, pathogenic Vibrio species that infect humans and marine organisms during warmer summer months are of growing concern. Coral reefs, in particular, are already experiencing unprecedented degradation worldwide...
- Genetic control of susceptibility to infection with Mycobacterium tuberculosis in mice. Mitsos, L-M; Cardon, L R; Fortin, A; Ryan, L; LaCourse, R; North, R J; Gros, P // Genes & Immunity;Dec2000, Vol. 1 Issue 8, p467
Genetic factors play a key role in host response, disease severity, and ultimate outcome of infection with Mycobacterium tuberculosis in humans. In the mouse, the DBA/2J strain is very susceptible to M. tuberculosis H37Rv infection, while the C57BI/6J strain is resistant. In DBA/2J, a heavier...
- Immune Mechanisms Underlying Host Susceptibility to Infection with Group A Streptococci. Goldmann, Oliver; Chhatwal, Gursharan Singh; Medina, Eva // Journal of Infectious Diseases;3/1/2003, Vol. 187 Issue 5, p854
Different strains of mice differ markedly in their susceptibility to group A streptococci (GAS) infection, with BALB/c mice being much more resistant than C3H/HeN mice. In this study, we examined the mechanisms underlying host resistance/susceptibility to GAS infection in these 2 mouse strains....
- The corallivorous invertebrate Drupella aids in transmission of brown band disease on the Great Barrier Reef. Nicolet, K.; Hoogenboom, M.; Gardiner, N.; Pratchett, M.; Willis, B. // Coral Reefs;Jun2013, Vol. 32 Issue 2, p585
Brown band disease (BrB) is an increasingly prevalent coral disease in the Indo-Pacific, but although the macroscopic signs of BrB have been associated with the ciliate Philaster guamensis, many aspects of its ecology remain unknown, particularly how the disease is transmitted among coral...
- Genetic analysis of innate resistance to mouse cytomegalovirus (MCMV). Beutler, Bruce; Georgel, Philippe; Rutschmann, Sophie; Zhengfan Jiang; Croker, Ben; Crozat, Karine // Briefings in Functional Genomics & Proteomics;Nov2005, Vol. 4 Issue 3, p203
Innate immunity is inherited and is, therefore, particularly susceptible to analysis by classical genetic methods. The 'phenotype first' approach has already revealed the principal receptors of the innate immune system as well as several essential signalling intermediates. It has recently...
- Why don't pigs get diabetes? Explanations for variations in diabetes susceptibility in human populations living in a diabetogenic environment. Gerstein, Hertzel C.; Waltman, Laura // CMAJ: Canadian Medical Association Journal;1/3/2006, Vol. 174 Issue 1, p25
This article examines variations in diabetes susceptibility in human populations living in a diabetogenic environment. It is inferred that susceptibility and resistance to diabetes may be related to environmental pressures. These pressures suggest that metabolic consequences of a diabetogenic...
- The Diabetes Type 1 Locus Idd6 Modulates Activity of CD4+ CD25+ Regulatory T-Cells. Rogner, Ute Christine; Lepault, Fran�oise; Gagnerault, Marie-Claude; Vallois, David; Morin, Jo�lle; Avner, Philip; Boitard, Christian // Diabetes;Jan2006, Vol. 55 Issue 1, p186
The genetic locus Idd6 confers susceptibility to the spontaneous development of type 1 diabetes in the NOD mouse. Our studies on disease resistance of the congenic mouse strain NOD.C3H 6.VIII showed that Idd6 influences T-cell activities in the peripheral immune system and suggest that a major...
