TITLE

The facilitator in acute kidney injury: TWEAK

AUTHOR(S)
Sanz, A. B.; Sánchez Sánchez-Nino, M. D.; Izquierdo, M. C.; Moreno, J. A.; Ucero, A. C.; Benito-Martín, A.; Santamaría, B.; Burgos, C.; Egido, J.; Ramos, A.; Berzal, S.; Coto, E.; Ruiz-Ortega, M.; Blanco-Colio, L. M.; Ortiz, A.
PUB. DATE
December 2008
SOURCE
Nefrologia;Dec2008, Vol. 28 Issue 6, p587
SOURCE TYPE
Academic Journal
DOC. TYPE
Article
ABSTRACT
The article presents information on acute kidney injury (AKI). It is informed that acute and chronic failure of renal function is characterized by loss of renal tubular cells. Tubular cell death is believed to be the best histopathological correlate of renal dysfunction during AKI. A study is being presented which attempts to identify factors which contribute to AKI. The study involves a novel cytokine-ligand pair, Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) and Fn14 gene.
ACCESSION #
37364960

 

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