Callahan, Arthur B.; Lutz, Brenton R.; Fulton, George P.; Degelman, John
February 1960
Angiology;Feb1960 Part 1, Vol. 11 Issue 1, p35
Academic Journal
1. A method for determining the smooth muscle threshold, using unipolar stimulation, of small arterioles (20 μ to 100 μ) is reported. 2. A method for determining the thrombus threshold of small blood vessels (20 μ to 100 μ), using unipolar stimulation is also reported. 3. The values obtained are summarized in table 1, and the physiologic factors concerned are discussed.


Related Articles

  • Endothelial Injury Induces Vascular Smooth Muscle Cell Proliferation in Highly Localized Regions of a Direct Contact Co-culture System. Joyce Wong // Cell Biochemistry & Biophysics;Sep2008, Vol. 52 Issue 1, p37 

    Abstract  Though previous studies have indicated a relationship between the proliferation of endothelial cells and vascular smooth muscle cells (VSMCs) in co-culture, the results have been contradictory and the signaling mechanism poorly understood. In this transmembrane co-culture study,...

  • Caveolins in vascular smooth muscle: Form organizing function. Hardin, Christopher D.; Vallejo, Johana // Cardiovascular Research;Mar2006, Vol. 69 Issue 4, p808 

    Abstract: Caveolae are becoming increasingly recognized as an important organizational structure for a variety of signal and energy-transducing systems in vascular smooth muscle (VSM). In this review, we discuss the emerging role of the caveolins in organizing and modulating the basic functions...

  • Matrix metalloproteinases regulate migration, proliferation, and death of vascular smooth muscle cells by degrading matrix and non-matrix substrates. Newby, Andrew C. // Cardiovascular Research;Feb2006, Vol. 69 Issue 3, p614 

    Abstract: Intimal thickening occurs in blood vessels in response to injury or atherosclerosis. The balance of migration and proliferation of vascular smooth muscle cells (VSMC) over death by apoptosis has an important impact on the final size of intimal thickening and may also affect...

  • Protection of Endothelial Cells, Inhibition of Neointimal Hyperplasia by �-elemene in an Injured Artery. Lingyan Wu; Guixue Wang; Shutang Tang; Guang Long; Tieying Yin // Cardiovascular Drugs & Therapy;Jun2011, Vol. 25 Issue 3, p233 

    ims: It is generally accepted that the oxidative stress and the proliferative activity of vascular smooth muscle cells (VSMCs) contribute to the pathogenesis of neointimal hyperplasia after vascular injury. Although �-elemene (�-1-methyl-1-vinyl-2, 4-diisopropenyl-cyclohexane) has been...

  • Modulation of thiopental-induced vascular relaxation and contraction by perivascular adipose tissue and endothelium. Kassam, S. I.; Lu, C.; Buckley, N.; Gao, Y. J.; Lee, R. M. K. W. // BJA: The British Journal of Anaesthesia;Aug2012, Vol. 109 Issue 2, p177 

    Background Thiopental induces relaxation of vascular smooth muscle cells through its direct and/or indirect vasodilator effects. The perivascular adipose tissue (PVAT) and the endothelium are known to attenuate vascular contraction, and we have recently reported that PVAT potentiates the...

  • Role of constitutively expressed heme oxygenase-2 in the regulation of guinea pig coronary artery tone. Gagov, Hristo; Kadinov, Boris; Hristov, Kiril; Boev, Kiril; Itzev, Dimiter; Bolton, Thomas; Duridanova, Dessislava // Pflugers Archiv European Journal of Physiology;Jul2003, Vol. 446 Issue 4, p412 

    Carbon monoxide (CO) is well known as a relaxing substance in the vasculature, where it is released during the heme oxygenase (HO) reaction. Little is known about the tissue-specific targets of CO in smooth muscles. To date the functional role of CO in the coronary artery remains unclear. The...

  • The glomerular cell: Source and target of various mediators. Briner, Verena A.; Pfeilschifter, Josef // Kidney International Supplement;Oct1997, Issue 61, pS-45 

    Reports on the balance between endothelium-derived mediators and the subsequent response in adjacent vascular smooth muscle cells (VSMC) and VSMC-like glomerular mesangial cells (GMC). Capacity of the mediators to regulation migration and proliferation of VSMC and GMC; Provision of tools to...

  • Local Regulation of Digital Blood Flow in Generalized Scleroderma. Kristensen, Johannes K. // Journal of Investigative Dermatology;May79, Vol. 72 Issue 5, p235 

    A decreasing reactivity of the vascular bed upon changes in vascular transmural pressure and changes in arterial perfusion pressure head was observed from band to fingers and from subcutis to cutis in patients suffering from generalized scleroderma. These findings are expressions of...

  • Sympathetically evoked Ca2+ signaling in arterial smooth muscle. Wei-jin Zang; Zacharia, Joseph; Lamont, Christine; Wier, Withrow Gil // Acta Pharmacologica Sinica;Dec2006, Vol. 27 Issue 12, p1515 

    The sympathetic nervous system plays an essential role in the control of total peripheral vascular resistance and blood flow, by controlling the contraction of small arteries. Perivascular sympathetic nerves release ATP, norepinephrine (NE) and neuropeptide Y. This review summarizes our...


Read the Article


Sorry, but this item is not currently available from your library.

Try another library?
Sign out of this library

Other Topics