TITLE

INJURY AND REPAIR OF ARTERIAL TISSUE

AUTHOR(S)
Lindler, J.
PUB. DATE
November 1974
SOURCE
Angiology;Nov1974, Vol. 25 Issue 10, p628
SOURCE TYPE
Academic Journal
DOC. TYPE
Article
ABSTRACT
It appears from the biochemical, histochemical and morphological analyses under discussion in this session, that the polysaccharide-protein components of the arterial ground substance, containing hexosamine, uronic acids, neuraminic acid etc. (acid and neutral proteoglycans) determine essential fundamental processes of atherosclerosis as well as every physiological and pathological metabolism in the vessel wall. So we have to understand (and further to investigate) arterial injuries and repair. The changes in the arterial fibre components depend on the chromotropic ground substance and its particular changes. We have discussed the special role of endothelia and of the dominating arterial cell, the smooth muscle cell. Our results (papers, discussions and references) further show summarizing, that atherosclerosis starts and developes with an increase of anabolism and catabolism. The particular findings of the disturbed metabolism of the intima and media in certain stages of injured arteries, especially in atherosclerosis, are demonstrated in details. The early increased anabolism and catabolism decrease under the normal level in the later atherosclerotic stages, the catabolic processes more and earlier than the anabolic processes. It results in the atherosclerotic insufficiency of the vessel wall metabolism with the well-known disorganisation, transformation and destruction, with lipid and calcium depositions in arteries. As has been stated, however, in spite of the numerous researches, aided by a diversity of methods used for the identification of the many groups of substances and components involved and their manifold interrelationships, these processes cannot yet be properly understood in detail, nor correlated with each other significantly until now. That will be the task of our further investigations on �Injury and Fiepair in Arterial Tissue�.
ACCESSION #
16357849

 

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