Immortal longings
Tags: STEM cells -- Therapeutic use; AGING -- Genetic aspects; TELOMERE; TELOMERASE; CELL death -- Prevention; PHYSIOLOGY
Related Articles
- Disguise cells to fix damaged tissue. Geddes, Linda // New Scientist;1/7/2012, Vol. 213 Issue 2846, p14
The article reports on research published in "Blood" by scientist Jeffrey Karp and team into the engineering of mesenchymal stem cells (MSCs) with an immune cell molecule that allows them to be transported to damaged tissues for healing.
- Fountain of Youth? // Scientist;Apr2011, Vol. 25 Issue 4, p24
An interview with InnerSpace Foundation founder Preston Estep regarding the role of telomeres in the aging process is presented. Estep says that telomere attrition is most critical in highly proliferative tissue and is not the sole cause of senescence. He states that telomerase elongation...
- Telomere Length, Telomerase, and Aging. Warner, Huber R.; Hodes, Richard J. // Generations;Spring2000, Vol. 24 Issue 1, p48
Examines the role of telomere length in aging and cancer. Hype and hope surrounding the issue of anti-aging possibilities; Reason for the shortening of telomeres; Effect of telomerase deficiency in humans.
- Amorcyte Patent Filing a First for Cardiac Stem Cell Therapy. Shaffer, Catherine // BioWorld Today;9/15/2010, Vol. 21 Issue 178, p1
The article reports on the issuance of the first patent for a stem cell therapy to treat heart damage following a heart attack by the U.S. Patent and Trademark Office to Amorcyte Inc. Patent number 7,794,705 was given to the AMR-001 product after Amorcyte confirmed a mechanism of action in its...
- Mayo Clinic Health Letter Oct 2011 Highlights: Managing the Side Effects of Chemotherapy; Using Stem Cells for Healing; Choosing Omega-3s for Heart Health. // Biomedical Market Newsletter;10/26/2011, Vol. 21, p33
The article presents the highlights of the October 2011 issue of "Mayo Clinic Health Letter." It offers tips on the benefits of chemotherapy and in managing its side effects such as eating well, enough rest, moderate exercise. It notes on the early stages of regenerative medicine, specifically...
- Discovery of a novel imprinted gene by transcriptional analysis of parthenogenetic embryonic stem cells. Sritanaudomchai, Hathaitip; Ma, Hong; Clepper, Lisa; Gokhale, Sumita; Bogan, Randy; Hennebold, Jon; Wolf, Don; Mitalipov, Shoukhrat // Human Reproduction;Aug2010, Vol. 25 Issue 8, p1927
BACKGROUND: Parthenogenetic embryonic stem cells (PESCs) may have future utilities in cell replacement therapies since they are closely related to the female from which the activated oocyte was obtained. Furthermore, the avoidance of parthenogenetic development in mammals provides the most...
- Adult Stem Cell Transplantation in Stroke: Its Limitations and Prospects. Jae-Kyu Roh; Keun-Hwa Jung; Kon Chu // Current Stem Cell Research & Therapy;Sep2008, Vol. 3 Issue 3, p185
A growing number of studies have demonstrated stem cell-based therapy provides a feasible, realistic approach to the restoration of lost brain function after stroke. Moreover, adult stem cells may provide more appropriate clinical strategies. Leading candidate sources include bone marrow,...
- Fountain of youth? Stover, Dawn // Popular Science;Feb99, Vol. 254 Issue 2, p57
Reports on biomedical researchers' discovery of a way to prevent human cells from aging. Gene that causes cells to produce an enzyme called telomerase; Presence of telomerase in the majority of all cancers; Possible uses of telomerase to control both aging and cancer; Cells' event counter which...
- Telomerase prevents the accelerated cell ageing of Werner syndrome fibroblasts. Wyllie, Fiona S.; Jones, Christopher J.; Skinner, Julia W.; Haughton, Michele F.; Wallis, Corrin; Wynford-Thomas, David; Faragher, Richard G.A.; Kipling, David // Nature Genetics;Jan2000, Vol. 24 Issue 1, p16
Werner syndrome (WS) is a rare disorder inherited in an autosomal recessive manner and characterized by accelerated ageing. WS fibroblasts display an accelerated rate of senescence in vitro, which has been linked to this progeroid phenotype. The senescence of normal human fibroblasts is...
