TITLE

An attack on aging

AUTHOR(S)
Gorman, Christine
PUB. DATE
January 1998
SOURCE
Time;1/26/1998, Vol. 151 Issue 3, p60
SOURCE TYPE
Periodical
DOC. TYPE
Article
ABSTRACT
Points out how scientists have discovered how an enzyme called telomere can revive dying cells. The experiment, which is sponsored by the US National Institute of Aging and the Geron biotech company of Menlo Park, California, as appearing in the journal `Science'; The evidence that each of the 1 trillion cells in the human body contains its own biological clock, which tells the cell when to stop growing and start dying; The theory as to why the cell plans obsolescence.
ACCESSION #
121726

 

Related Articles

  • An attack on aging. Gorman, Christine // Time International (South Pacific Edition);01/26/98, Issue 4, p53 

    Points out how scientists have discovered how an enzyme called telomerase can revive dying cells. The experiment, which is sponsored by the US National Institute on Aging and the Geron biotech company of Menlo Park, California, as appearing in the journal `Science'; The evidence that each of...

  • The Man Who Would Stop Time. HOOPER, JOSEPH // Popular Science;Aug2011, Vol. 279 Issue 2, p50 

    The article looks a Bill Andrews, a molecular biologist who is seeking a way to prevent aging on the molecular level. His research centers on telomeres, the sequences of DNA bases which assist with cell division and are shortened each time a cell replicates, and his company, Sierra Sciences,...

  • Immortal longings. Klotzko, Arlene Judith // New Scientist;12/05/98, Vol. 160 Issue 2163, p49 

    Focuses on possible therapeutic uses of embryonic or stem cells. The ability of stem cells to repair and reverse the effects of aging; The differentiation that stem cells are capable of; The activity of telomerase in stem cells; The role that telomerase plays in keeping telomeres active; The...

  • Preventing cells from committing suicide.  // USA Today Magazine;Jun98, Vol. 126 Issue 2637, p4 

    Focuses on the prevention of apoptosis or programmed cell death. Why human cells commit suicide; Advantage of apoptosis; Factor that triggers cellular suicide.

  • Suicidal brain cells can be saved. Knight, Jonathan // New Scientist;05/09/98, Vol. 158 Issue 2133, p21 

    Reports that a way has been found to reduce the damage to brain cells temporarily deprived of oxygen. Discovery that brain cells do not die as soon as they are deprived of oxygen; Length of time that cells can survive without oxygen; Description of apoptosis, or cell suicide; Enzymes called...

  • Neuroprotection not yet a clinical reality. Charters, Lynda; Katz, L. Jay // Ophthalmology Times;06/15/2001, Vol. 26 Issue 12, p62 

    Provides information on neuroprotection, a technique in cell death prevention. Application of neuroprotection in preventing glaucomatous damage; Purpose of neuroprotection in glaucoma; Mechanism of action of some neuroprotective agents.

  • Glutathione depletion-induced apoptosis of Ha-ras-transformed NIH3T3 cells can be prevented by melatonin. Chuang, Jih I; Chang, Tsuey Y; Liu, Hsiao S // Oncogene;3/6/2003, Vol. 22 Issue 9, p1349 

    It is well known that intracellular antioxidant glutathione (GSH) plays major roles in the maintenance of redox status and defense of oxidative stress. Ras, a small GTP-binding protein, may send growth-stimulating message to the nucleus through downstream Rac oncoprotein and superoxide (O2-)....

  • Inhibition of type IIA secretory phopholipase A2 reduces cell-death of cardiomyocytes in acute myocardial infarction in rats. Vermond, R.A.; Van Dijk, A.; Krijnen, P.A.J.; Spreeuwenberg, M.D.; Visser, F.C.; Van Milligen, F.J.; Niessen, H.W.M. // European Journal of Heart Failure. Supplements;Jun2008, Vol. 7 Issue 1, p139 

    An abstract of the article "Inhibition of type IIA secretory phopholipase A2 reduces cell-death of cardiomyocytes in acute myocardial infarction in rats," by R. A. Vermond and colleagues is presented.

  • Cell death: NonALKylating activity. Donner, Amy // Nature Chemical Biology;Jul2013, Vol. 9 Issue 7, p408 

    The article offers information on the study which revealed that ALKBH7's stable genetic knockdown presents resistance to cell death.

Share

Read the Article

Courtesy of THE LIBRARY OF VIRGINIA

Sorry, but this item is not currently available from your library.

Try another library?
Sign out of this library

Other Topics