Radiation oncology

August 1997
Clinical & Investigative Medicine;Aug97 Supplement, Vol. 20, pS81
Academic Journal
Presents an abstract of the research manuscript `Repopulation kinetics during multifractional irradiation in an in vitro tumor model,' by E. Sham and R.E. Durand. Cell kinetics.


Related Articles

  • Targeting αvβ3 and αvβ5 inhibits photon-induced hypermigration of malignant glioma cells.  // Radiation Oncology;2011, Vol. 6 Issue 1, p132 

    The article reports on a study conducted to analyze the modifiability of photon-altered glioma cell motility by integrin inhibition. The study would also describe the mechanisms underlying increased glioma cell migration through photon irradiation. The study results revealed that peripheral...

  • Ionizing radiation induces a motile phenotype in human carcinoma cells in vitro through hyperactivation of the TGF-beta signaling pathway. Carl, Cedric; Flindt, Anne; Hartmann, Julian; Dahlke, Markus; Rades, Dirk; Dunst, Jürgen; Lehnert, Hendrik; Gieseler, Frank; Ungefroren, Hendrik // Cellular & Molecular Life Sciences;Jan2016, Vol. 73 Issue 2, p427 

    Radiotherapy, a major treatment modality against cancer, can lead to secondary malignancies but it is uncertain as to whether tumor cells that survive ionizing radiation (IR) treatment undergo epithelial-mesenchymal transition (EMT) and eventually become invasive or metastatic. Here, we have...

  • How molecular motors work. Spudich, James A. // Nature;12/8/1994, Vol. 372 Issue 6506, p515 

    Discusses the molecular basis of cell movement and changes in cell shape. Review of related literature; Integration of approaches to support to the mechanochemical actin-activated myosin ATPbase cycle; Roles for surface loops.

  • Erratum.  // Journal of Clinical Investigation;Nov2010, Vol. 120 Issue 11, p4163 

    A correction to the article "Lack of Cell Movement Links Four Developmental Disorders," that was published in the previous issue is presented.

  • Protective Bystander Effects Simulated with the State-Vector Model. Sch�llnberger, Helmut; Eckl, Peter M. // Dose-Response;2007, Vol. 5 Issue 3, p187 

    Apoptosis induced in non-hit bystander cells is an important biological mechanism which operates after exposure to low doses of low-LET radiation. This process was implemented into a deterministic multistage model for in vitro neoplastic transformation: the State-Vector Model (SVM). The new...

  • A Role for Bioelectric Effects in the Induction of Bystander Signals by Ionizing Radiation? Mothersill, C.; Moran, G.; McNeill, F.; Gow, M. D.; Denbeigh, J.; Prestwich, W.; Seymour, C. B. // Dose-Response;2007, Vol. 5 Issue 3, p214 

    The induction of "bystander effects" i.e. effects in cells which have not received an ionizing radiation track, is now accepted but the mechanisms are not completely clear. Bystander effects following high and low LET radiation exposure are accepted but mechanisms are still not understood. There...

  • Models of cell locomotion. Baumgaertner, A.; Sambeth, R. // AIP Conference Proceedings;2000, Vol. 501 Issue 1, p112 

    The motion of single biological cells is a fascinating phenomena which has puzzled scientists for many decades. The current physical concepts of cell motility are reviewed and the recent progress of our understanding concerning the role of polymerization processes are summarized. In particular,...

  • Glioblastoma: Looking at the Currently Marketed Sigma-1 Agonists and Antagonists. Kast, Richard E. // Neoplasia;Aug2007, Vol. 9 Issue 8, p689 

    A letter to the editor is presented in response to an article about the role of sigma-1 agonist 4-IBP [1] in the reduction of the motility of glioblastoma cells.

  • Side glance: Pseudopodia.  // JNCI: Journal of the National Cancer Institute;3/15/95, Vol. 87 Issue 6, p409 

    Presents a photograph of a cancer cell's pseudopod or false foot. Function of pseudopods in movement.


Read the Article


Sorry, but this item is not currently available from your library.

Try another library?
Sign out of this library

Other Topics